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At therapeutic doses, the small amount of NAPQI produced is neutralized by conjugation with reduced glutathione (GSH), as shown in Figure 1, a crucial tripeptide that, in an NADPH-dependent reaction, helps detoxify oxidants like NAPQI

Moreover, APAP-induced mitochondrial damage has been shown to partly result from the suppression of PPAR-regulated pathways, leading to irreversible inhibition of fatty acid oxidation

SOD2, superoxide dismutase 2

Nature 458:762765 Galan-Cobo A, Sitthideatphaiboon P, Qu X et al (2019) LKB1 and KEAP1/NRF2 Pathways cooperatively promote metabolic reprogramming with enhanced glutamine dependence in KRAS-mutant lung adenocarcinoma

In the heart, H2 receptors are essential in controlling the rhythm